COVID-19’s Biological Politics

Gallaher-Garry Trajectory, continued,

From post #1,839, we mentioned Chaetomium for onychomycosis.

Chaetomium
'....Many cases cause type 1 allergic reactions and infections. A few cases of fatal deep infections due to C. atrobrunneum have been reported in immunocompromised people. Other clinical syndromes include brain abscess, peritonitis, and onychomycosis.'

The peritonitis caused by Chaetomium links to feline infectious peritonitis coronavirus (FIPV), which if not mistaken, also infects brushtail opposum for E.C. Holmes's Australia.

Next, before linking FIPV to UC Davis, California, we show the Grave's disease link to the RGD motif of Parkinson's disease.
 
RFK Jr.'s book due date out has been moved forward to 18 Ap 2023. There is no doubt changes being made up the the last minute to counter media propaganda and other fascist pathologies. Twitter censoring is a current hot topic:
 
The Gallaher-Garry collaboration linking Grave's disease to a feline leukemia retrovirus is compared with Grave's disease carrier protein, because we are searching for the furin cleavage site link to feline infectious peritonitis virus, and because we have already aligned Gallaher as the first Westerner to mouth off about the FCS in Chinese SARS2, an amino acid sequence that was already found on Long Island well before the pandemic, well before Gallaher's limelighting, in Ap 2013.

Gallaher-Garry Grave's disease links to ectoparasites of animals and humans, some of which take blood meals, such as the Long Island Ixodes scapularis Lyme disease tick that harbored and RGD sequence (2008), a sequence also found in SARS2.

Human Body Louse, Pediculus humanus subsp. corporis / Grave's Disease Carrier Protein
 
We trust that the reader is already familiar with who Yong-Zhen Zhang is: the first human to sequence SARS2.

The next Grave's disease carrier protein, now having been linked to Gallaheer-Garry, is now linked to Yong-Zhen Zhang's crony, co-author and sidekick, Guan-Zhu Han for plant signaling against pathogens, already posted to USMB:

Hibiscus syriacus Grave's Disease Carrier Protein / Furin Cleavage Site
'Grave's disease carrier protein: 'RARR.'

The Hibiscus RARR is a motif recognized by furin (USMB search: RXXR). There has now been posted evidence for both the SARS2 RGD motif and the SARS2 furin cleavage site (FCS), one from ticks and one from plants, respectively, linking four H. sapiens: Gallaher-Garry <---->Zhang-Han
 
We posted this earlier (post # 1,801) and Washburne et al go into some detail here:

H5N1 Classic Spillover vs. SARS2
 
Adding the military connection to Hunter Biden's Metabiota...

Human Coronavirus 229E / Cameroon
'....Hipposideros ruber, anal swab, coll. 30 May 2018, Cameroon....Military Health Research Center (CRESAR); Mosaic; UC Davis; Metabiota; Global Viral.'

This virus carries the RGD motif as does the Chinese military's (PLA) virus, ZC45 (Dr. Quay's Twitter page, 24 Aug 2022).
 
Readers can where breakfast_dogs highlights the 15 repeats of HKU1, beginning with NDDEDV, which functions are unknown. Also, the receptor was unknown, at least up to May 2017, though there is resonance with influenza and HIV-1 in this regard (O-acetylation of sialic acid, galectin, respectively).

The original discovery paper's authors include Leo Poon, Rosana W. Poon, thus linking to SARS2 as well.

It should be mentioned HKU1n Patient Zero's co-morbidities:
'....71 year old, smoker, history of pulmonary tuberculosis, COPD, Pseudomonas aeruginosa colonization of airways, hyperlipidemia, asymptomatic abdominal aortic aneurysm.'
 
Earlier, RFK Jr. mentions ACIP:
Post #1,769

Currently, a video that includes ACIP is here:
 
C-19 Theological Techniques



The violence of esoterica is exploited whereby the patient's "vaccination" status remains esoterica, for eternity, while the patient is given no chance to change the violence of the regulation, being kept from family members as they die, which violence will affect further investigations into the origin of the infecting virus itself.
 
This mutation is linked to the Elf, as will be shown, though it is by default linked to Baric's North Carolina lab:

S:F456L Mutation Growing Fastly in North Carolina
'Corneliusroemer commented last week: 'If it turns out to be beneficial, it would represent an interesting 2-step move of XBB: first increase ACE2 binding affinity through S486P, then trading that increased affinity for immune escape.

It's not surprising that 456L (S:F456L mutation) has not been seen much until now as the ACE2 binding decrease is rather large, making this mutation only survivable to viruses with already high ACE2 affinity. 456L appeared in some Delta lineages (AY.122) just before Omicron arrived....S:F456L apparently growing fastly in USA (North Carolina).'

Roemer misses the history of this F-to-L mutation with regard to Ralph Baric's recombinants, paradoxically, produced in North Carolina. In addition, this mutation links to The Elf.
 

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