Fully vaxxed fake native American Elizabeth Warren tests positive for Covid

Is the elf doing prophylaxis or reacting to PCR tests? If it's the latter, it's already too late. The commie virus in deer will mutate differently than in the African mice that evolved Omicron.
African Mice ... eh Sherlock ? Are they the same pesky critters that make a
living out of the insides of junked cars.Mostly cars not trucks.
Mice have preferences also.No respectable mouse would be seen within a
3 blocks of Biden's White House.
 
African Mice ... eh Sherlock ? Are they the same pesky critters that make a
living out of the insides of junked cars.Mostly cars not trucks.
Mice have preferences also.No respectable mouse would be seen within a
3 blocks of Biden's White House.
Dipshit, you're living proof that you cannot read science posted to USMB. You are making a fool of yourself in front of the others that can read and comprehend the science we have posted about that.
 
Badger understands that the concept is dangerously misleading, being projected by an authoritarian nazi-dem-controlled media. Viruses don't stop being dangerous just because it makes a nazi feel uncomfortable. They are evolving with impunity, outside any vaccine parameters, and the CDC is clamming up about them. The prisoners need to be made aware of this alternate evolution of the commie virus that got here for free and doesn't require application for American citizenship.
Despite your paranoid lunacy, "fully vaccinated" still means as many shots as needed to be at maximum efficacy, not 100% protection.
 
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The reason we ask is because, no doubt about it, Omicron mutation N969K is a vaccine-linked mutation.
Who's "we?"

And no, Omicron is not a vaccine-linked mutation. Share this among your "we" crowd....

FACT: COVID-19 vaccines do not create or cause variants of the virus that causes COVID-19. Instead, COVID-19 vaccines can help prevent new variants from emerging.​
New variants of a virus happen because the virus that causes COVID-19 constantly changes through a natural ongoing process of mutation (change). As the virus spreads, it has more opportunities to change. High vaccination coverage in a population reduces the spread of the virus and helps prevent new variants from emerging. CDC recommends that everyone 5 years of age and older get vaccinated as soon as possible.​
Learn more about variants.​
 
Despite your paranoid lunacy, "fully vaccinated" still means as many shots as needed to be at maximum efficacy, not 100% protection.
Stupids such as yourself will be the first to deny they've been duped. You don't get to define "fully vaxxed" as your ass sees it with impunity to critique. Because you're not educated enough in science to be discussing it, you try to hide the absurdity of this accelerated low-quality Jesus of the vaccine mafia by projecting the blm-like violence of your lack of education onto what you think are convenient scapegoats.

Dipshit, neither does the CDC care to debate the vaccine-linked Omicron mutation, N969K, but the nazi-dem effeminates who control the media will say both the U.S./ Army and Pfizer are working to defeat Omicron. Another absurdity is that this dumbing-down by the nazi-dems include the fact that Omicron evolved in African mice first, not African men. F you just the same, there are SARS-CoV-2 viruses mutating and freely traveling in the environment without ever confronting the vaccine.
 
Who's "we?"

And no, Omicron is not a vaccine-linked mutation. Share this among your "we" crowd....

FACT: COVID-19 vaccines do not create or cause variants of the virus that causes COVID-19. Instead, COVID-19 vaccines can help prevent new variants from emerging.​
New variants of a virus happen because the virus that causes COVID-19 constantly changes through a natural ongoing process of mutation (change). As the virus spreads, it has more opportunities to change. High vaccination coverage in a population reduces the spread of the virus and helps prevent new variants from emerging. CDC recommends that everyone 5 years of age and older get vaccinated as soon as possible.​
Learn more about variants.​
Not if an entirely different population of the commie virus is mutating and evolving in a non-human host such as Omicron did in African mice, then (jumping back [itaclis]) into humans. Dipshit, "we" are all of the authors in badger's head being respected by always citing them when we can, so "we" does more to show your stupidity and lack of science education than you may care to admit. F you just the same.
 
Immune systems at five years of age are not mature and some systems may not be mature unti 12 years of age. This is high pedophiliac roulette on the part of the medical mafia. Which vaccine do they get shot-up with? Chimp-based? A non-coronavirus with Chinese commie parts thrown in? Boycott the Pimp.
 
Who's "we?"

And no, Omicron is not a vaccine-linked mutation. Share this among your "we" crowd....

FACT: COVID-19 vaccines do not create or cause variants of the virus that causes COVID-19. Instead, COVID-19 vaccines can help prevent new variants from emerging.​
New variants of a virus happen because the virus that causes COVID-19 constantly changes through a natural ongoing process of mutation (change). As the virus spreads, it has more opportunities to change. High vaccination coverage in a population reduces the spread of the virus and helps prevent new variants from emerging. CDC recommends that everyone 5 years of age and older get vaccinated as soon as possible.​
Learn more about variants.​
What you are not fully knowledgeable of, is as to whether or not the changes are changes into a more dangerous strain or into a less dangerous strain ?? So until you have complete knowledge of this, then you are just causing fear about something you don't even understand yourself. So what is it then, are you politically motivated or scientifically motivated ?? It maybe that you are politically motivated, otherwise just projecting because your handler's have you doing such projecting.

Go back to the 1918 pandemic, and study how that virus finally disappeared. Was it by vaccine's or by natural immunity overcoming it finally ?

Aug 19, 2008 (CIDRAP News) A study of the blood of older people who survived the 1918 influenza pandemic reveals that antibodies to the strain have lasted a lifetime and can perhaps be engineered to protect future generations against similar strains.

The findings appeared online Aug 17 in Nature. Study collaborators hail from several institutions: Vanderbilt University, Mount Sinai School of Medicine, University of Medicine and Dentistry of New Jersey (UMDNJ), the Centers for Disease Control and Prevention (CDC), and Scripps Research Institute.

Inspiration for the study came from an unlikely source, an episode of an old medical television show that portrayed a town protecting itself from the 1918 virus outbreak by using blood from an elderly survivor, the Associated Press (AP) reported yesterday. The storyline prompted Eric Altschuler, MD, a professor of physical medicine and rehabilitation at UMDND to ask the National Institutes of Health (NIH) for a grant to test people over age 90 for the 1918 flu antibodies, according to the AP report. The NIH funded much of the study and enlisted the expertise of other experts.

The group collected blood samples from 32 pandemic survivors aged 91 to 101. The multipronged study had four components, to:

  • Determine if the survivors still had antibodies to the virus
  • See if the B cellsthe ones that produce the antibodiescould be cultured and produce antibodies to a 1918 virus protein
  • Attempt fusing cells having the highest levels of activity with myeloma cells to create a hybrid cell line that secretes monoclonal antibodies
  • Evaluate if the antibodies could protect mice infected with the 1918 influenza virus
The people recruited for the study were 2 to 12 years old in 1918 and many recalled sick family members in their households, which suggests they were directly exposed to the virus, the authors report. The group found that 100% of the subjects had serum-neutralizing activity against the 1918 virus and 94% showed serologic reactivity to the 1918 hemagglutinin.

The investigators generated B lymphoblastic cell lines from the peripheral blood mononuclear cells of eight subjects. Transformed cells from the blood of 7 of the 8 donors yielded secreting antibodies that bound the 1918 hemagglutinin.

Author James E. Crowe, Jr, MD, professor of pediatrics and director of the Vanderbilt Program in Vaccine Sciences, said in a press release from Vanderbilt that the researchers were surprised by the findings.

"The B cells have been waiting for at least 60 yearsif not 90 yearsfor that flu to come around again," he said. "That's amazing, because it's the longest memory anyone's ever demonstrated."

From the B cells of three donors, the research group generated five monoclonal antibodies that not only strongly neutralized the 1918 virus, but also cross-reacted with proteins related to the 1930 swine flu virus. However, the antibodies did not react against more contemporary influenza strains.

In the final arm of the study, the researchers infected mice with the reconstructed 1918 virus and the next day tested the five monoclonal antibodies at various doses to see if the therapy protected the animals. The mice receiving the lowest dose of the 1918 monoclonal antibody died, as did the ones receiving the control antibody. All given the highest antibody doses survived.

Dr Tshidi Tsibane, a study author and postdoctoral fellow in Mount Sinai School of Medicine's microbiology department, said in a press release from Mount Sinai that though there is no need for a new treatment for 1918 influenza virus infections, the results are still useful.

"These findings could serve as potential therapy for another 1918-like virus," said Dr Tsibane in the statement.

The authors point out that it is difficult to be certain that the monoclonal antibodies they isolated were first stimulated during the 1918 influenza pandemic. However, they write that the subjects' clinical histories and the high affinity of the monoclonal antibodies for the 1918 strain "strongly suggest that recent exposures do not account for this immunity." They add that exposure to similar viruses circulating during first part of the 20th century probably bolstered the subjects' B cell function.

Anthony Fauci, MD, director of the National Institute of Allergy and Infectious Diseases, said recent studies have projected that immunity lasts several decades; the current study provides proof, the AP reported. "This is the mother of all immunological memory here," he told the AP.
 
Who's "we?"

And no, Omicron is not a vaccine-linked mutation. Share this among your "we" crowd....

FACT: COVID-19 vaccines do not create or cause variants of the virus that causes COVID-19. Instead, COVID-19 vaccines can help prevent new variants from emerging.​
New variants of a virus happen because the virus that causes COVID-19 constantly changes through a natural ongoing process of mutation (change). As the virus spreads, it has more opportunities to change. High vaccination coverage in a population reduces the spread of the virus and helps prevent new variants from emerging. CDC recommends that everyone 5 years of age and older get vaccinated as soon as possible.​
Learn more about variants.​
You are mistaken. Omicron mutation N969K is a vacccine-linked mutation. We (all these other authors) would be glad to debate this fact with anyone. You were duped like the rest of the prisoners in not
knowing the difference between a variant and a mutation.
 
What you are not fully knowledgeable of, is as to whether or not the changes are changes into a more dangerous strain or into a less dangerous strain ?? So until you have complete knowledge of this, then you are just causing fear about something you don't even understand yourself. So what is it then, are you politically motivated or scientifically motivated ?? It maybe that you are politically motivated, otherwise just projecting because your handler's have you doing such projecting.

Go back to the 1918 pandemic, and study how that virus finally disappeared. Was it by vaccine's or by natural immunity overcoming it finally ?

Aug 19, 2008 (CIDRAP News) A study of the blood of older people who survived the 1918 influenza pandemic reveals that antibodies to the strain have lasted a lifetime and can perhaps be engineered to protect future generations against similar strains.

The findings appeared online Aug 17 in Nature. Study collaborators hail from several institutions: Vanderbilt University, Mount Sinai School of Medicine, University of Medicine and Dentistry of New Jersey (UMDNJ), the Centers for Disease Control and Prevention (CDC), and Scripps Research Institute.

Inspiration for the study came from an unlikely source, an episode of an old medical television show that portrayed a town protecting itself from the 1918 virus outbreak by using blood from an elderly survivor, the Associated Press (AP) reported yesterday. The storyline prompted Eric Altschuler, MD, a professor of physical medicine and rehabilitation at UMDND to ask the National Institutes of Health (NIH) for a grant to test people over age 90 for the 1918 flu antibodies, according to the AP report. The NIH funded much of the study and enlisted the expertise of other experts.

The group collected blood samples from 32 pandemic survivors aged 91 to 101. The multipronged study had four components, to:

  • Determine if the survivors still had antibodies to the virus
  • See if the B cellsthe ones that produce the antibodiescould be cultured and produce antibodies to a 1918 virus protein
  • Attempt fusing cells having the highest levels of activity with myeloma cells to create a hybrid cell line that secretes monoclonal antibodies
  • Evaluate if the antibodies could protect mice infected with the 1918 influenza virus
The people recruited for the study were 2 to 12 years old in 1918 and many recalled sick family members in their households, which suggests they were directly exposed to the virus, the authors report. The group found that 100% of the subjects had serum-neutralizing activity against the 1918 virus and 94% showed serologic reactivity to the 1918 hemagglutinin.

The investigators generated B lymphoblastic cell lines from the peripheral blood mononuclear cells of eight subjects. Transformed cells from the blood of 7 of the 8 donors yielded secreting antibodies that bound the 1918 hemagglutinin.

Author James E. Crowe, Jr, MD, professor of pediatrics and director of the Vanderbilt Program in Vaccine Sciences, said in a press release from Vanderbilt that the researchers were surprised by the findings.

"The B cells have been waiting for at least 60 yearsif not 90 yearsfor that flu to come around again," he said. "That's amazing, because it's the longest memory anyone's ever demonstrated."

From the B cells of three donors, the research group generated five monoclonal antibodies that not only strongly neutralized the 1918 virus, but also cross-reacted with proteins related to the 1930 swine flu virus. However, the antibodies did not react against more contemporary influenza strains.

In the final arm of the study, the researchers infected mice with the reconstructed 1918 virus and the next day tested the five monoclonal antibodies at various doses to see if the therapy protected the animals. The mice receiving the lowest dose of the 1918 monoclonal antibody died, as did the ones receiving the control antibody. All given the highest antibody doses survived.

Dr Tshidi Tsibane, a study author and postdoctoral fellow in Mount Sinai School of Medicine's microbiology department, said in a press release from Mount Sinai that though there is no need for a new treatment for 1918 influenza virus infections, the results are still useful.

"These findings could serve as potential therapy for another 1918-like virus," said Dr Tsibane in the statement.

The authors point out that it is difficult to be certain that the monoclonal antibodies they isolated were first stimulated during the 1918 influenza pandemic. However, they write that the subjects' clinical histories and the high affinity of the monoclonal antibodies for the 1918 strain "strongly suggest that recent exposures do not account for this immunity." They add that exposure to similar viruses circulating during first part of the 20th century probably bolstered the subjects' B cell function.

Anthony Fauci, MD, director of the National Institute of Allergy and Infectious Diseases, said recent studies have projected that immunity lasts several decades; the current study provides proof, the AP reported. "This is the mother of all immunological memory here," he told the AP.
But Fau Chi is not coming clean here, when he says that. Fau is relying on most not to know about the coronavirus link to this lasting immunity. The 1930 swine flu hemagglutinin most definitely links to PHEV coronavirus (porcine hemagglutinating encephalomyeltitis virus), a unique virus that links to influenza. That SARS-CoV-2 jumped into South African mice to evolve Omicron is Fau's worst nightmare, because it has the potential to make vaccines out of style. We have yet to compare amino acid sequences of PHEV with Omicron. mutations.
 
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